MYELOID NEOPLASIA Novel imatinib-sensitive PDGFRA-activating point mutations in hypereosinophilic syndrome induce growth factor independence and leukemia-like disease

نویسندگان

  • Christian Elling
  • Philipp Erben
  • Christoph Walz
  • Marie Frickenhaus
  • Mirle Schemionek
  • Martin Stehling
  • Hubert Serve
  • Nicholas C. P. Cross
  • Andreas Hochhaus
  • Wolf-Karsten Hofmann
  • Wolfgang E. Berdel
  • Carsten Müller-Tidow
چکیده

1Department of Medicine A, Hematology, Oncology, and Pneumology, University of Münster, Münster, Germany; 2III Medizinische Klinik, Universitätsmedizin Mannheim, Universität Heidelberg, Mannheim, Germany; 3Max-Planck-Institute for Molecular Biomedicine, Münster, Germany; 4Department of Medicine, Hematology, and Oncology, University of Frankfurt, Frankfurt, Germany; 5Human Genetics Division, University of Southampton School of Medicine, Southampton, United Kingdom; and 6Abteilung Hämatologie/Onkologie, Universitätsklinikum Jena, Jena, Germany

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Novel imatinib-sensitive PDGFRA-activating point mutations in hypereosinophilic syndrome induce growth factor independence and leukemia-like disease.

The FIP1L1-PDGFRA fusion is seen in a fraction of cases with a presumptive diagnosis of hypereosinophilic syndrome (HES). However, because most HES patients lack FIP1L1-PDGFRA, we studied whether they harbor activating mutations of the PDGFRA gene. Sequencing of 87 FIP1L1-PDGFRA-negative HES patients revealed several novel PDGFRA point mutations (R481G, L507P, I562M, H570R, H650Q, N659S, L705P,...

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تاریخ انتشار 2011